dilated cardiomyopathy
di·lat·ed car·di·o·my·op·a·thy
dilated cardiomyopathy
Cardiology The most common cardiomyopathy in the US, which is usually idiopathic and characterized by ↑ ventricular size and impaired ventricular function Etiology Infection–eg coxsackievirus, CMV, HIV, diphtheria, trichinosis, inflammation–eg connective tissue disease, sarcoidosis, metabolic–eg hypothyroidism, thyrotoxicosis, DM, Cushing's disease, thiamine, selenium deficiency, or toxic–eg cocaine, antiretroviral agents, lead, cobalt, ethanol, phenothiazines insults; ±20% of DCs, there is a familial component Prognosis Often progresses to CHF accompanied by mitral and tricuspid valve insufficiency Complications Conduction defects–eg atrial & ventricular tachyarrhythmias and fibrillation Management Supportive–eg rest, weight control, smoking cessation, ↓ physical activity during exacerbation; therapies that may be effective include ACE inhibitors, anticoagulation, digoxin, diuretics, implantable defibillators, nitrates, potassium, magnesium repletion Investigational modalities Amiodarone, amlodipine, beta-blockers, dual-chamber pacing, felodipine, pimobendan, vesnarinone; recombinant hGH has been reported to ↑ myocardial mass and ↓ left ventricular chamber size, resulting in improved hemodynamics, myocardial energy metabolism, clinical statusBeck·er dis·ease
(bek'ĕr di-zēz')Synonym(s): dilated cardiomyopathy.